· 김승현, 노학재. (2002). 신경-근 접합부 질환에서의 전기생리검사. 대한임상신경생리학회 지, 4(2), 171-188.
· 이동국. (1999). 신경전도검사의기초기술. 대한임상신경생리학회지, 1(2), 202-209.
· 식품의약품안전처 www.mfds.go.kr
· Dawson DM, Potts F. (1991). Acute nontraumatic myelopathies. Neurol Clin, 9(3), 585-603.
· Fontaine B. (2008). Periodic paralysis. Adv Genet, 63, 3-23.
· Holland OB, Brown H, Kuhnert L, Fairchild C, Risk M, Gomez-Sanchez CE. (1984). Further evaluation of saline infusion for the diagnosis of primary aldosteronism. Hypertension, 6(5), 717-723.
· John W. Funder, Robert M. Carey, Franco Mantero, M. Hassan Murad, Martin Reincke, Hirotaka Shibata, Michael Stowasser, and William F. Young, Jr (2016). The Management of Primary Aldosteronism: Case Detection, Diagnosis, and Treatment: An Endocrine Society Clinical Practice Guideline. J Clin Endocrinol Metab, 101(5), 1889–1916.
· Liu T, Nagami GT, Everett ML, Levine BS. (2005). Very low calorie diets and hypokalaemia: the importance of ammonium excretion. Nephrol Dial Transplant, 20(3), 642-646.
· Massey JM. (1997). Acquired myasthenia gravis. Neurol Clin, 15(3), 577-595.
· Mattsson C, Young WF Jr. (2006). Primary aldosteronism: diagnostic and treatment strategies. Nat Clin Pract Nephrol, 2(4), 198-208.
· Patterson JR, Grabois M. (1986). Locked-in syndrome: a review of 139 cases. Stroke, 17(4), 758-764.
· Ribstein J, Du Cailar G, Fesler P, Mimran A. (2005). Relative glomerular hyperfiltration in primary aldosteronism. J Am Soc Nephrol., 16(5), 1320-1325.
· Rosow LK, Amato AA. (2016). The Role of Electrodiagnostic Testing, Imaging, and Muscle Biopsy in the Investigation of Muscle Disease. Continuum (Minneap Minn), 22(6), 1787-1802.
· Sawka AM, Young WF, Thompson GB, Grant CS, Farley DR, Leibson C, van Heerden JA. (2001). Primary aldosteronism: factors associated with normalization of blood pressure after surgery. Ann Intern Med., 135(4), 258-261.
· Sebastian A, McSherry E, Morris RC Jr . (2010). On the mechanism of renal potassium wasting in renal tubular acidosis associated with the Fanconi syndrome (type 2 RTA). J Clin Invest., 50(1), 231-243.
· Serdar O., Selcuk Y., Lacin T., Yasemin G.,, Tayfun G., and Ali T. (2013). Hypokalemia- induced myopathy and massive creatine kinase elevation as first manifestation of Conn's syndrome. Niger Med J., 54(4), 283–284.
· Stryjewski TP, Papakostas TD, Vavvas D. (2016). Proliferative Hypertensive Retinopathy. JAMA Ophthalmol, 134(3), 345-346.
· Stowasser M, Sharman J, Leano R, Gordon RD, Ward G, Cowley D, Marwick TH. (2005). Evidence for abnormal left ventricular structure and function in normotensive individuals with familial hyperaldosteronism type I. J Clin Endocrinol Metab., 90(9), 5070-5076.
· Weisbrod AB, Webb RC, Mathur A, Barak S, Abraham SB, Nilubol N, Quezado M, Stratakis CA, Kebebew E. Adrenal histologic findings show no difference in clinical presentation and outcome in primary hyperaldosteronism. Ann Surg Oncol., 20(3), 753-758.
· Willison HJ, Jacobs BC, van Doorn PA. (2016). Guillain-Barrésyndrome. Lancet, 388(1), 717-727.
· Young WF (2007). Primary aldosteronism: renaissance of a syndrome. Clin Endocrinol (Oxf), 66(5), 607-618.