고혈압전단계자에 대한 유산소운동이 심혈관반응에 미치는 영향 (Cardiovascular Responses in Prehypertensive Individuals during Dynamic Exercise)

서지정보

· 발행기관 : 한국사회체육학회
· 수록지 정보 : 한국사회체육학회지 / 42호 / 1049 ~ 1056페이지
· 저자명 : 홍선주, 김종경, 최현민, 박시영, 이준희, 전종목, 노호성

초록

본 연구는 일상생활에서 운동습관이 없는 18~26세 남자대학생을 정상혈압자 10명과 고혈압전단계자 10명으로 분류하여 안정 시와 운동 시 심혈관반응을 검토하였다.
최근에 개발된 non-invasive CO 장비(Physio flow)를 이용하여, 고혈압전단계자가 운동 시 과도한 심혈관반응이 유발된다는 사실을 본 연구는 처음으로 시도되었다. 중앙명령과 동맥압력반사기능과 상관없이 운동 시 운동승압반사에 의한 과도한 혈압반응과 CO반응이 유발된다는 성과를 얻게 되었다. 이러한 과도한 혈압반응은 말초혈관 저항의 증가 때문이 아니라 CO의 증가 때문에 일어났다. 이와 같이 고혈압전단계나 고혈압에서 운동 시 심혈관반응을 조절하는데 운동승압반사의 기능이 중요한 역할을 하는 것으로 나타났다.

영어초록

It is generally known that in hypertensive individuals, exercise induces exaggerated increase in arterial blood pressure and in turn this dangerous elevation may increase the risks for myocardial infarction, cardiac arrest, or stroke. However, it has still not been investigated whether the exaggerated cardiovascular responses occur in prehypertension and these excessive responses are mediated by exercise pressor reflex overactivity. Therefore,the purpose of this study was to examine the effects of exercise pressor reflex on the cardiovascular responses when exercise intensity increased from rest through low exercise intensity to high exercise intensity. Twenty young, fit people (10 normotension and 10 prehypertension) with normal body fat percentage were recruited for this study. All subjects were instrumented to measure the cardiovascular responses at rest and during exercise. A catheter to collect blood samples was inserted into left brachial vein. Systolic blood pressure,diastolic blood pressure, and mean arterial blood pressure were significantly higher in prehypertension across workloads compared to the normotension. Stroke volume (SV), heart rate (HR), and cardiac output (CO) were significantly increased in both groups with increasing workloads, but SV and CO were significantly higher in prehypertension. There was no difference in total vascular conductance in both groups. Thus, exaggerated blood pressure responses were mediated via increases in CO. This study suggests that the excessive cardiovascular responses to exercise in prehypertension may be due to dysfunction of exercise pressor reflex.

참고자료

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