효소측정의 임상적 응용

등록일 1999.02.19 한글 (hwp) | 5페이지 | 무료

본문내용

Mechanism for abnormal serum enzyme level
Increased level
Increased release
Necrosis : Myocardial infarction(AST, LD, CK, HBDH)
Acute Hepatitis(AST, ALT, OCT LD, LAP)
Acute pancreatitis(Amylase, Lipase)
Increased permeability without necrosis
: Progressive muscular dystrophy(CK, ALS, LD)
Increased tissue source of enzyme
: Neoplastic disease (carcinoma, lymphoma, leukemia)(LD, ALS)
Megaloblastic anemia(LD, ALS)
Osteoblastic lesion(Paget's disease, osteogenic sarcoma, rickets,
healing fracture)(ALP)
Impaired excretion of enzyme
: Uremia (Pepsinogen, Amylase)
Obstructive jaundice( ALP, LAP, r-GT, 5'-N)
Decreased level
Decreased formation
Genetic : Hypophosphatasia(ALP)
Wilson's disease(Ceruloplasmin)
Acholinesterasemia(Pseudocholinesterase)
Acquired : Hepatitis(Pseudocholinesterase)
Starvation(Amylase)
Enzyme ingibition
: Insecticide poisoning(Pseudocholinesterase)
Lack of cofactors
: Pregnancy(AST)
Cirrhosis

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